The chemokine MCP-1 (CCL2), which contributes to monocyte recruitment to sites of infection, was significantly higher at all time points in the propofol-treated groups, as were the eosinophil chemoattractant eotaxin and the neutrophil chemoattractant KC (CXCL1) (Fig 3A). The gene discussed is CXCL1; the disease is infection.