CD24 and cancer: Interestingly, miR-17 inhibition resulted in 2.4-fold and 1.96-fold increase in the number of colonies formed in Caki-1 and ACHN, respectively, demonstrating capabilities of inducing self-renewal and significantly increased expression for mesenchymal markers such as ZEB1, ZEB2, vimentin, and N-cadherin and for cancer stem markers CD24 and CD44 [22].