Following CLP-sepsis, the time-dependent disruption of pulmonary microvascular/PMVEC barrier function, as reflected by pulmonary microvascular EB-albumin permeability in vivo, was strongly correlated (r = 0.976, p < 0.05; Fig. 3a) with IVVM PMVEC death (PI+). The gene discussed is ALB; the disease is Sepsis.