Application of the same approach to the Smarcb1 dependent phosphorylation under serum starvation revealed that peptides phosphorylated in Smarcb1 deficient tumor cells are enriched for targets of AKT, a result consistent with our previous findings demonstrating activation of AKT in the same Smarcb1 deficient cell line used for the phosphoproteomic analysis. Here, SMARCB1 is linked to neoplasm.