CLEC7A and fungal infectious disease: As we could not find any differences in these cell populations in naive mice, this indicates that Dectin-1 is important for the maintenance of DC populations during pathogenic fungal infection but is unaffected by commensal microbes, including fungi, in the gut.8 Indeed, these data are consistent with our previous observation showing that Dectin-1 expression is not detected on the luminal-facing side of the intestinal barrier,21 and that this receptor is not involved in controlling commensal populations of C. albicans in the GI tract.14