TP53 and endometrial cancer: In endometrial cancer, oncogenes such as p53 gain-of-function mutations [21], KLF17 [79], EZH2, MCL-1 and FOS [8] promote EMT-associated invasiveness and enhance CSC properties including self-renewal capacity and chemoresistance, whereas miR-101, miR-106b, miR-130b and miR-194 [7, 8, 22, 80] serve as EMT suppressors and attenuate CSC features.