ALK and anaplastic large cell lymphoma: Since autophagy inhibition (either by pharmacological inhibition or by an siATG7-mediated approach) potentiates the anti-tumoral activity of ALK inactivation (either by crizotinib treatment or by an siALK-mediated approach), our results indicate that autophagy could act as a survival mechanism in therapeutically-challenged ALK-positive ALCL cells.