Very recently, a growing number of papers on SFT have been published, describing an NAB2-STAT6 fusion gene in the tumor [20–22, 24, 33], the diagnostic relevance of STAT6 or GRIA2 IHC [4, 5, 25, 26], relationships between fusional variations of the NAB2-STAT6 gene, histological features and clinical prognosis [23, 24, 30], and expression profiles of genes other than NAB2-STAT6 [24, 34]. Here, STAT6 is linked to neoplasm.