Animal experiments indicate that IFN-ɤ-deficient mice are more susceptible to S. aureus infections compared to WT mice [36,38], suggesting that the Th1 cellular response is important in providing protection against S. aureus. Moreover, IL-17-producing Th17 cells are important for fighting S. aureus infection by recruiting neutrophils to the site of infection [38]. The gene discussed is IFNA1; the disease is infection.