Additionally, Schulte el at by screening the subcellular distribution of Htt labeled by mRFP and monitoring the morphology of cultured neurons via live-imaging discovered 18b-glycyrrhetinic acid, as well as camptothecins, OH-camptothecin, carbenoxolone inhibited formation of Htt aggregate, restored neurite morphology and viability in Drosophila HD model [55]. Here, HTT is linked to Huntington disease.