In the study mentioned above (by Mockenhaupt et al) which was carried out among a similar study population (pregnant women attending antenatal clinic in the Ashanti region of Ghana)[14], where the genotypes were determined, homozygous deficiency prevalence (2.6%) was similar to the 2.3% G6PD full deficiency in this study but the prevalence of heterozygous deficiency (30.4%) was quite different. The gene discussed is G6PD; the disease is hyperinsulinemic hypoglycemia, familial, 4.