Genes encoding IκBα/β/ε (which sequester NF-κB in the cytoplasm) and IκB-zeta (which inhibits NF-κB transcription activity) were significantly upregulated in either GCB- or ABC-DLBCL (Supplementary Fig. S3M-P), resembling p65 function [38]. The gene discussed is NFKB1; the disease is aneurysmal bone cyst.