Gocyk et al. showed that gastric H. pylori infection in lung cancer patients is accompanied by a significant increase in gastrin plasma and bronchial lavage levels as well as by increased mRNA expression for gastrin and its receptors, as well as for Cyclo oxygenase-1 (COX-1) and COX-2 in the tumor tissue [14]. The gene discussed is GAST; the disease is neoplasm.