Because both recovery in KCNQ currents and reduction in HCN channel activity are associated with resilience to tinnitus, we hypothesized that either KCNQ plasticity occurs before HCN channel plasticity and drives the resilience pathway or that plasticity in HCN channels occurs before KCNQ channel plasticity and plays the determinant role for tinnitus resilience. Here, MALAT1 is linked to Tinnitus.