Given that NF-κB mediates cell survival [9,10] as well as transcriptional upregulation and synthesis of the assayed cytokines [2,13], it was hypothesized that inhibition of NF-κB in EMT-6 cells would improve PDT efficacy and reduce the pro-inflammatory signaling by PDT-afflicted tumor cells. Here, NFKB1 is linked to neoplasm.