In keeping with the capability of miRNAs from the miR-17-92 cluster to regulate specific BCR pathway genes, DLBCLs characterized by higher levels of many components of the BCR signalling cascade (e.g. CD19, IG, CD79a, BLK, SYK, PLCγ2, and MAP4K) has been found to express higher MYC and miR-17-92 transcript levels than other DLBCL subtypes [54, 69]. Here, MYC is linked to diffuse large B-cell lymphoma.