However, a recent study reported that encephalomyocarditis virus (EMCV) has the ability to disrupt SGs by cleaving G3BP1 in order to avoid the innate immune detection of its infection and subsequent induction of IFN-β (Ng et al., 2013), suggesting that another effective strategy for viral evasion from the IFN system by preventing avSG formation exists. Here, IFNA1 is linked to infection.