Since very little is known about human–Aspergillus interactions in available databases up to date, we selected the infection-relevant subnetwork of interactions between the host GO slim term “symbiosis, encompassing mutualism through parasitism” and the fungal GO slim term “pathogenesis.” To get a transparent size, we visualized only host nodes pathogenicity-associated based on HPIDB and removed the human interactor UBC (ubiquitin C) due to the high number of interactions. This evidence concerns the gene UBC and infection.