Moreover, other groups have focused on modulation of the cell adhesion function by GalNAc-T3 overexpression, which is involved in invasion/metastasis of the ovarian adenocarcinoma via abnormal glycosylation of O-glycoprotein mucin 1 (MUC1), potentially accompanied by the downregulated expression of E-cadherin and β-catenin [24]. Here, MUC1 is linked to ovarian adenocarcinoma.