However, in the present case, despitecontinuous treatment with GCV and the inclusion of foscarnet in the therapeutic regimen,HCMV load remained relatively stable (Figure 2).During follow-up at the AIDS Clinic, the patient developed bilateral HCMV retinitis(Figure 1) that was non-responsive to GCV andwas only influenced by foscarnet administration and an increase in the CD4+cell count. This evidence concerns the gene CD4 and retinitis.