STAT5A and neoplasm: We were also able to validate the increase in both STAT3 and STAT5a, GNB2L1/RACK1, PTEN, and PARP (all validated in Fig. 10C) in xenograft-derived tumor lysates overexpressing MOAP-1, although the receptor for the STATs, Janus kinase 1 (JAK1), did not appear to be up-regulated (confirmed in Fig. 10C).