Current evidence, which suggests a pathologic role for SCA17 with low expansions, is mostly based on case descriptions showing movement disorders or psychiatric symptoms accompanied by CAG expansions in TBP.[10–13] Because the statistical analysis between the normal controls and patients with low expansion repeats failed to show any differences so far, we must consider that clinical cases with low expansion repeats could be idiopathic movement disorders showing coincidental CAG/CAA expansions. The gene discussed is TBP; the disease is movement disorder.