CRP and coronary artery disorder: Data on association between the variants at CRP locus and the coding of amino acids are currently limited and +942G>C (rs1800947) was previously reported to exert no influence on the amino acid.14,15 Several lines of evidence have established a significant connection of genetic variants with CRP levels, including +942G>C, −717A>G (rs2794521), and +1444C>T (rs1130864).16,17 The functional properties of these variants prompted a number of investigators to evaluate their contributions to genetic risk of CAD.