10 recently demonstrated an association between low levels of CD4+FOXP3+ cells and increased risk of myocardial infarction indicating that Tregs are also atheroprotective in humans. In line with this, a reduction in the number of Tregs has been reported in patients with non‐ST elevation acute coronary syndrome (NSTE‐ACS) or unstable angina, whereas levels in patients with stable coronary artery disease (CAD) were similar to those in healthy subjects 11, 12, 13. The gene discussed is CD4; the disease is coronary artery disorder.