Although the combination of GAD antibody–related limb stiffness, dysautonomia, and CIP appeared to be unique, the literature does provide case examples for each of the above symptoms with autoimmune origin to respond to intravenous immunoglobin (IvIg).3-5 We offered this therapy to our patient at intervals of 4 weeks. The gene discussed is GAD1; the disease is hereditary sensory and autonomic neuropathy.