As this scenario appears to be more related to CSF‐1‐dependent tumor‐associated macrophages that drive proliferation and dedifferentiation of mutant (tumor) cells (Mantovani and Sica, 2010) than to macrophage‐related processes going on in GBS/AMAN, it is a tempting to speculate that pathogenic mechanism within the nerve can strongly vary among the different nerve disorders but can show clear commonalities with disorders not at all related to the nervous system (e.g., cancer). This evidence concerns the gene CSF1 and neoplasm.