As such, the biological associations to hereditary and neurological diseases and disorders among genes containing overlapping CAML genotypes in the NTC/NCC lineage cancers suggests that modification to non-coding, intronic regions are (1) sensitive to disease manifestation and, (2) differing combinations of tissue-specific CAML genotypes may contribute to diseases of NTC/NCC lineage, albeit different disease phenotypes, due to alternative splicing via non-coding variants [37]. This evidence concerns the gene CAMLG and nervous system disorder.