The increased presence of macrophages combined with elevated levels of various chemokines and cytokines such as MCP-1 (monocyte chemotactic protein-1), TNF-α, and oncostatin M during the pathogenesis of dilated cardiomyopathy underscores the contribution of cardiac inflammation [6, 15, 24–26]. This evidence concerns the gene CCL2 and dilated cardiomyopathy.