An imbalance between an angiogenic agent (vascular endothelial growth factor [VEGF]) and an antiangiogenic factor (soluble fms-like tyrosine kinase-1 [sFlt-1]) is seen in women with preeclampsia [22–24], twin pregnancy [24], and PPCM [24], and an antiangiogenic environment is seen in these three conditions with elevated levels of sFlt-1 [22–24]. Here, VEGFA is linked to preeclampsia.