The fraction of the MLCP regulatory subunit MYPT which was phosphorylated at the inhibitory Thr855 site (i.e. the proportion of MLCP in the putative RhoA/ROK-dependent inactive state) was not different in unstimulated platelets taken from either non-septic shock or septic shock patients (6.482 ± 1.32 vs. 7.658 ± 1.07; n = 22 to 24 per patient group; Figure 6). The gene discussed is RHOA; the disease is septic shock.