In fact, among the several known “oncomiRs” overexpressed in our samples, we found miR-106b and miR-93, recently shown to target NKG2DL, a ligand of the activating receptor of natural killer (NK) cells NKG2D, and thus suggested to contribute to the immune evasion typical of glioblastoma cells [56]. Here, KLRK1 is linked to glioblastoma.