In the gastric cancer patients, there was no significant difference in the distribution of the STAT3 genotypes and the number of EPIYA-C segments (p = 0.16; Kruskal Wallis test) and cagA status (χ2 = 2.73, 2 degrees of freedom, p = 0.26) (Table 3). Here, S100A8 is linked to gastric cancer.