GPAT2 and Insulin resistance: In order to assess other mechanisms involved in the pathogenesis of hepatic insulin resistance, we evaluated the triglyceride content and the expression of molecules that participate in lipogenesis in the liver, such as sterol-regulatory element binding protein-1 (Srebp-1), stearoyl coenzyme A desaturase-1 (Scd-1), acetyl-CoA carboxylase (Acc), fatty acid synthase (Fas) and glycerol-3-phosphate acyltransferase 2 (Gpat2).