It has also been described that CD14+HLA-DRlow/− monocytes isolated from PCa patients expressed high level of IL-10, inhibited autologous T cell proliferation more effectively than (CD14+HLA-DR+) monocytes from healthy individuals, and were defective in their ability to differentiate into phenotypically mature DCs [45]. The gene discussed is CD14; the disease is posterior cortical atrophy.