AKT2 and prostate cancer: Enhanced expression of miR-29b in metastatic prostate cancer cells accordingly reverses EMT and inhibits the invasive phenotype,27 whereas miR-30a expression is reduced during transforming growth factor-β-induced EMT in murine hepatocytes.28 According to our results, miR-612 suppressed CRC metastasis by inhibiting AKT2, which is also a contributor to EMT.