AMH and polycystic ovary syndrome: As AMH is produced in granulosa cells of growing follicles [23], these findings suggest that, considering still ongoing excessive follicle recruitment (i.e., still high AMH levels), non-obese PCOS patients at older ages produce relatively deficient amounts of T in ovarian theca cells and/or adrenals, even though in young controls with NFOR, these T levels would be considered in entirely normal range.