Some research showed that mutations in SLC34A2 caused Pulmonary Alveolar Microlithiasis (PAM) [7] and anomalous expression of SLC34A2 was responsible for some other diseases such as hypophosphatemia, infertility and Testicular Microlithiasis (TM) [7, 8]. This evidence concerns the gene SLC34A2 and hypophosphatemia.