BCL2L2 and glioblastoma: To verify the above data, we examined whether miR-29b regulated the migratory potential, invasiveness, angiogenesis, and stemness maintenance of GBM by repressing BCL2L2. Thus, U251 and U87MG cells transfected with si-control, si-BCL2L2, anti-miR-29b, or co-transfected with si-BCL2L2 and anti-miR-29b were subjected to migration, invasion, and neurosphere formation assays (Figure 6A), and immunoblotting (Figure 6B).