Infection with live, wild‐type (WT) N. meningitidis leads to a marked increase in E‐selectin, Intracellular Adhesion Molecule‐1 (ICAM‐1) and Vascular Adhesion Molecule‐1 (VCAM‐1) endothelial expressions (Dixon et al., 2004). The gene discussed is SELE; the disease is infection.