Additionally, when glomerular filtration rate (GFR) in SCD patients was modeled as a function of the previously reported MYH9 risk haplotype and the APOL1 recessive model, we observed a significant interaction between the two genes, suggesting that APOL1 and MYH9 may act together to induce SCDN[5]. This evidence concerns the gene MYH9 and Schnyder corneal dystrophy.