We found that keratinocytes stimulated with PGN from S. aureus induce the overexpression of LL37 and VEGF (Figure 1, f, c and j), but downexpressed VHL (Figure 1, g) [94]; furthermore, when we transfected keratinocytes with ll-37 VEGF and IAP-2 they were highly expressed (Figure 1, j and e) suggesting that this antiapoptotic molecule could favor keratinocytes proliferation in psoriasis and could participate in the VEGF expression to promote angiogenesis [95]. This evidence concerns the gene CAMP and psoriasis.