Bindea and colleagues showed growth acceleration of MC38 isografts in Cxcr5−/− mice (CXCR5 is the receptor for CXCL13) compared to control, and rejection of tumour cells when recombinant CXCL13 was injected into the colonic submucosa of wild‐type mice before transplantation 96. The gene discussed is CXCL13; the disease is neoplasm.