Wu et al. [162] demonstrated that CAPE inhibits in vitro and in vivo MCF-7 and MDA-MB-231 tumour growth without much effect on normal mammary cells by reducing the expression of growth and transcription factors, including NF-κB. Recently, it was demonstrated that CAPE effect on genes that are associated with tumour cell growth and survival is related in part to its role as a histone deacetylase inhibitor [163]. Here, NFKB1 is linked to neoplasm.