KCNJ2 and myocardial infarction: Taken together, our results indicate that CK2 is a potentially important regulator of KCNJ2 gene expression and an important determinant of cardiac electronic instability following MI, via IK1. Additionally, our findings indicate that CK2 is a potential mediator of the electrophysiological effects of valsartan and provide a basis for the improvement of IK1 remolding facilitated by valsartan.