As for INH-induced hepatitis, 1 NAT2 variant (857G>A, corresponding to NAT2∗7, a slow acetylator genotype) was associated with increased risk, and the wild type allele NAT2∗4, a rapid acetylator genotype was associated with decreased risk of INH-induced HATT (Supplementary Table S1, http://links.lww.com/MD/A303). This evidence concerns the gene NAT2 and hepatitis A virus infection.