Based on our observation that percentages of T regulatory cells were not altered by infection with T. crassiceps and because T. crassiceps infection recruits AAMФs into the peritoneal cavity (approximately 35% of peritoneal exudate cells are F4/80+MR+Arg1+) and peritoneal adherent cells suppress T cell proliferation [28], we evaluated whether this population was able to influence the development of DSS-induced colitis. Here, NR3C2 is linked to infection.