Our study has some limitations: (1) it is a cross-sectional study, and hence causality inferences cannot be made; (2) it was done in a small, nonrandom sample of subjects; (3) we did not include HOMA-IR, which could explain the relationship of insulin resistance with MS and inflammatory markers; (4) finally, we only determined Lp-PLA2 activity instead of mass. The gene discussed is PLA2G7; the disease is myeloid sarcoma.