The activated form of Akt, phosphorylated Akt (p-Akt), may inhibit several proapoptotic factors including glycogen synthase kinase-3 beta (GSK-3 beta), fork-head homolog in rhabdomyosarcoma (FKHR), Bcl-2-associated death protein, and caspase-9; each of them may influence neuronal survival after stroke [25]. The gene discussed is GSK3B; the disease is stroke disorder.