Although the normal glucose and insulin levels observed in 3w SRD C3H mice (Fig. 6, F and G) support the notion that these mice do not develop insulin resistance on an SRD, the normoinsulinemic levels preclude a direct assessment of the role for Pparγ in insulin-stimulated enhancement of hepatic Cd36 expression in C3H mice. The gene discussed is CD36; the disease is Insulin resistance.