Taking these data together, we therefore assume that our observation on up-regulated CD9 and DHRS3 and PTPN7 expression in T2D monocytes can (next to the over-expression of miR-34c-5p) be taken as a sign that the circulating monocytes in T2D patients have an altered potential for adhesion, migration and differentiation into progeny, such as macrophages, dendritic cells and vascular support cells. This evidence concerns the gene CD9 and type 2 diabetes mellitus.